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We have found that p38 mitogen-activated protein kinase serves as an important mechanism through which GLP-1 modulates myocardial injury

Acknowledgments We thank Gabriela Furlan, Noelia Maldonado and Alejandra Romero (Cell Culture Facility, Luciana Reyna (Molecular Biology Facility), Paula Abadie and Santiago Boccardo (Cytometry Facility), Pilar Crespo (Microscopy Facility), Victoria Blanco and Raul Villarreal (Animal Care Facility), Sergio Oms, Ariel Frontera, Diego Luti, Walter Requena, Cecilia Noriega, and German Romero (BIOPROAL - Animal Production Facility) for their skillful technical assistance at the CIBICI-CONICET/ Department of Clinical Biochemistry, School of Chemistry Sciences, National University of Cordoba

Maldonado-Sanabria et al., 2024)

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Skipping too many days Copper peptides work through cumulative signaling

By inhibiting NNMT using agents such as 5-amino-1MQ, it's possible to enhance NAD+ levels and activate genes that promote metabolism